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Our study effectively treated OA via a synergistic strategy of anti-senescence and pro-chondrogenesis that may inform the treatment of OA and other degenerative joint diseases

At cellular level NO promotes muscle homeostasis by stimulating glucose transport during exercise trough activation of upstream signaling events leading to the translocation of glucose transporter GLUT4 at the cell surface [70]

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Studies have shown TB-500 accelerates wound closure, promotes angiogenesis in ischemic tissue, and supports neural regeneration